History of Wolff‐Parkinson‐White Syndrome
Identifieur interne : 000061 ( Main/Corpus ); précédent : 000060; suivant : 000062History of Wolff‐Parkinson‐White Syndrome
Auteurs : Melvin M. ScheinmanSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 2005-02.
English descriptors
- KwdEn :
Abstract
While Drs. Wolff, Parkinson, and White fully described the syndrome that bears their names in 1930, prior case reports had already described the essentials. Over the ensuing century this syndrome has captivated the interest of anatomists, clinical cardiologists, and cardiac surgeons. Stanley Kent described lateral muscular connections over the atrioventricular (AV) groove, which he felt were the normal AV connections. The normal AV connections were, however, clearly described by His and Tawara. True right‐sided AV connections were initially described by Wood et al., while Öhnell first described left free wall pathways. David Scherf is thought to be the first to describe our current understanding of the pathogenesis of the Wolff‐Parkinson‐White (WPW) syndrome in terms of a reentrant circuit involving both the AV node—His axis as well as the accessory pathway. This hypothesis was not universally accepted and many theories were applied to explain the clinical findings. The basics of our understandings were established by the brilliant work of Pick, Langendorf, and Katz who by using careful deductive analysis of ECGs were able to define the basic pathophysiological processes. Subsequently, Wellens and Durrer applied invasive electrical stimulation to the heart in order to confirm the pathophysiological processes. Sealy and his colleagues at Duke University Medical Center were the first to successfully surgically divide an accessory pathway and ushered in the modern area for curative therapy for these patients. Morady and Scheinman were the first to successfully ablate an accessory pathway (posteroseptal) using high‐energy direct‐current shocks. Subsequently, Jackman, Kuck, Morady, and a number of groups proved the remarkable safety and efficiency of catheter ablation for pathways in all locations using radiofrequency energy. More recently, Gallob et al. first described the gene responsible for a familial form of WPW. The current ability to cure patients with WPW is due to the splendid contributions of individuals from diverse disciplines from throughout the world.
Url:
DOI: 10.1111/j.1540-8159.2005.09461.x
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ISTEX:8AF4179AD7FF5D60F05D57E4B7788C05986FA9B3Le document en format XML
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Wolff, Parkinson, and White fully described the syndrome that bears their names in 1930, prior case reports had already described the essentials. Over the ensuing century this syndrome has captivated the interest of anatomists, clinical cardiologists, and cardiac surgeons. Stanley Kent described lateral muscular connections over the atrioventricular (AV) groove, which he felt were the normal AV connections. The normal AV connections were, however, clearly described by His and Tawara. True right‐sided AV connections were initially described by Wood et al., while Öhnell first described left free wall pathways. David Scherf is thought to be the first to describe our current understanding of the pathogenesis of the Wolff‐Parkinson‐White (WPW) syndrome in terms of a reentrant circuit involving both the AV node—His axis as well as the accessory pathway. This hypothesis was not universally accepted and many theories were applied to explain the clinical findings. The basics of our understandings were established by the brilliant work of Pick, Langendorf, and Katz who by using careful deductive analysis of ECGs were able to define the basic pathophysiological processes. Subsequently, Wellens and Durrer applied invasive electrical stimulation to the heart in order to confirm the pathophysiological processes. Sealy and his colleagues at Duke University Medical Center were the first to successfully surgically divide an accessory pathway and ushered in the modern area for curative therapy for these patients. Morady and Scheinman were the first to successfully ablate an accessory pathway (posteroseptal) using high‐energy direct‐current shocks. Subsequently, Jackman, Kuck, Morady, and a number of groups proved the remarkable safety and efficiency of catheter ablation for pathways in all locations using radiofrequency energy. More recently, Gallob et al. first described the gene responsible for a familial form of WPW. The current ability to cure patients with WPW is due to the splendid contributions of individuals from diverse disciplines from throughout the world.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>MELVIN M. SCHEINMAN</name><affiliations><json:string>University of California San Francisco, San Francisco, California</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>historical review</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Wolff‐Parkinson‐White syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>catheter ablation</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>cardiac electrosurgery</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>preexcitation</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>accessory pathways</value></json:item></subject><articleId><json:string>PACE9461</json:string></articleId><language><json:string>eng</json:string></language><abstract>While Drs. Wolff, Parkinson, and White fully described the syndrome that bears their names in 1930, prior case reports had already described the essentials. Over the ensuing century this syndrome has captivated the interest of anatomists, clinical cardiologists, and cardiac surgeons. Stanley Kent described lateral muscular connections over the atrioventricular (AV) groove, which he felt were the normal AV connections. The normal AV connections were, however, clearly described by His and Tawara. True right‐sided AV connections were initially described by Wood et al., while Öhnell first described left free wall pathways. David Scherf is thought to be the first to describe our current understanding of the pathogenesis of the Wolff‐Parkinson‐White (WPW) syndrome in terms of a reentrant circuit involving both the AV node—His axis as well as the accessory pathway. This hypothesis was not universally accepted and many theories were applied to explain the clinical findings. The basics of our understandings were established by the brilliant work of Pick, Langendorf, and Katz who by using careful deductive analysis of ECGs were able to define the basic pathophysiological processes. Subsequently, Wellens and Durrer applied invasive electrical stimulation to the heart in order to confirm the pathophysiological processes. Sealy and his colleagues at Duke University Medical Center were the first to successfully surgically divide an accessory pathway and ushered in the modern area for curative therapy for these patients. Morady and Scheinman were the first to successfully ablate an accessory pathway (posteroseptal) using high‐energy direct‐current shocks. Subsequently, Jackman, Kuck, Morady, and a number of groups proved the remarkable safety and efficiency of catheter ablation for pathways in all locations using radiofrequency energy. More recently, Gallob et al. first described the gene responsible for a familial form of WPW. 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Wolff, Parkinson, and White fully described the syndrome that bears their names in 1930, prior case reports had already described the essentials. Over the ensuing century this syndrome has captivated the interest of anatomists, clinical cardiologists, and cardiac surgeons. Stanley Kent described lateral muscular connections over the atrioventricular (AV) groove, which he felt were the normal AV connections. The normal AV connections were, however, clearly described by His and Tawara. True right‐sided AV connections were initially described by Wood et al., while Öhnell first described left free wall pathways. David Scherf is thought to be the first to describe our current understanding of the pathogenesis of the Wolff‐Parkinson‐White (WPW) syndrome in terms of a reentrant circuit involving both the AV node—His axis as well as the accessory pathway. This hypothesis was not universally accepted and many theories were applied to explain the clinical findings. The basics of our understandings were established by the brilliant work of Pick, Langendorf, and Katz who by using careful deductive analysis of ECGs were able to define the basic pathophysiological processes. Subsequently, Wellens and Durrer applied invasive electrical stimulation to the heart in order to confirm the pathophysiological processes. Sealy and his colleagues at Duke University Medical Center were the first to successfully surgically divide an accessory pathway and ushered in the modern area for curative therapy for these patients. Morady and Scheinman were the first to successfully ablate an accessory pathway (posteroseptal) using high‐energy direct‐current shocks. Subsequently, Jackman, Kuck, Morady, and a number of groups proved the remarkable safety and efficiency of catheter ablation for pathways in all locations using radiofrequency energy. More recently, Gallob et al. first described the gene responsible for a familial form of WPW. The current ability to cure patients with WPW is due to the splendid contributions of individuals from diverse disciplines from throughout the world.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>historical review</term></item><item><term>Wolff‐Parkinson‐White syndrome</term></item><item><term>catheter ablation</term></item><item><term>cardiac electrosurgery</term></item><item><term>preexcitation</term></item><item><term>accessory pathways</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2005-02">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/8AF4179AD7FF5D60F05D57E4B7788C05986FA9B3/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Science Inc</publisherName><publisherLoc> 350 Main Street , Malden , MA 02148‐5018 , USA and 9600 Garsington Road , Oxford OX4 2DQ , UK . </publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="02002"><doi origin="wiley">10.1111/pace.2005.28.issue-2</doi><numberingGroup><numbering type="journalVolume" number="28">28</numbering><numbering type="journalIssue" number="2">2</numbering></numberingGroup><coverDate startDate="2005-02">February 2005</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="11" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.2005.09461.x</doi><idGroup><id type="unit" value="PACE9461"></id><id type="supplier" value="pace9461"></id></idGroup><countGroup><count type="pageTotal" number="5"></count></countGroup><titleGroup><title type="tocHeading1"><i>HISTORY</i></title></titleGroup><eventGroup><event type="firstOnline" date="2005-01-28"></event><event type="publishedOnlineFinalForm" date="2005-01-28"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.4 mode:FullText source:FullText result:FullText" date="2010-03-29"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="152">152</numbering><numbering type="pageLast" number="156">156</numbering></numberingGroup><correspondenceTo>Address for reprints: Melvin M. Scheinman, M.D., University of California San Francisco, 500 Parnassus Avenue, Box 1354, San Francisco, CA 94143‐1354. Fax: (415) 476‐6260; e‐mail: <email>scheinman@medicine.ucsf.edu</email></correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE9461.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received July 21, 2004; revised August 1, 2004; accepted August 16, 2004.</unparsedEditorialHistory><countGroup><count type="figureTotal" number="1"></count><count type="tableTotal" number="0"></count><count type="formulaTotal" number="0"></count><count type="referenceTotal" number="53"></count><count type="linksCrossRef" number="47"></count></countGroup><titleGroup><title type="main">History of Wolff‐Parkinson‐White Syndrome</title><title type="shortAuthors">SCHEINMAN</title><title type="short">HISTORY OF WOLFF‐PARKINSON‐WHITE SYNDROME</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>MELVIN M.</givenNames><familyName>SCHEINMAN</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="US"><unparsedAffiliation>University of California San Francisco, San Francisco, California</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1"><i>historical review</i></keyword><keyword xml:id="k2"><i>Wolff‐Parkinson‐White syndrome</i></keyword><keyword xml:id="k3"><i>catheter ablation</i></keyword><keyword xml:id="k4"><i>cardiac electrosurgery</i></keyword><keyword xml:id="k5"><i>preexcitation</i></keyword><keyword xml:id="k6"><i>accessory pathways</i></keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><!-- SCHEINMAN, M.: History of Wolff-Parkinson-White Syndrome. 
<p> <i>While Drs. Wolff, Parkinson, and White fully described the syndrome that bears their names in 1930, prior case reports had already described the essentials. Over the ensuing century this syndrome has captivated the interest of anatomists, clinical cardiologists, and cardiac surgeons. Stanley Kent described lateral muscular connections over the atrioventricular (AV) groove, which he felt were the normal AV connections. The normal AV connections were, however, clearly described by His and Tawara. True right‐sided AV connections were initially described by Wood et al., while Öhnell first described left free wall pathways. David Scherf is thought to be the first to describe our current understanding of the pathogenesis of the Wolff‐Parkinson‐White (WPW) syndrome in terms of a reentrant circuit involving both the AV node—His axis as well as the accessory pathway. This hypothesis was not universally accepted and many theories were applied to explain the clinical findings. The basics of our understandings were established by the brilliant work of Pick, Langendorf, and Katz who by using careful deductive analysis of ECGs were able to define the basic pathophysiological processes. Subsequently, Wellens and Durrer applied invasive electrical stimulation to the heart in order to confirm the pathophysiological processes. Sealy and his colleagues at Duke University Medical Center were the first to successfully surgically divide an accessory pathway and ushered in the modern area for curative therapy for these patients. Morady and Scheinman were the first to successfully ablate an accessory pathway (posteroseptal) using high‐energy direct‐current shocks. Subsequently, Jackman, Kuck, Morady, and a number of groups proved the remarkable safety and efficiency of catheter ablation for pathways in all locations using radiofrequency energy. More recently, Gallob et al. first described the gene responsible for a familial form of WPW. The current ability to cure patients with WPW is due to the splendid contributions of individuals from diverse disciplines from throughout the world</i>.</p><!-- (PACE 2005; 28:152–156)
</abstractGroup></contentMeta><noteGroup><note xml:id="n-fnt-1" numbered="no"><p>Dr. Scheinman would like to acknowledge research grant support from the Erin McEowen Memorial Fund.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>History of Wolff‐Parkinson‐White Syndrome</title></titleInfo><titleInfo type="abbreviated"><title>HISTORY OF WOLFF‐PARKINSON‐WHITE SYNDROME</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>History of Wolff‐Parkinson‐White Syndrome</title></titleInfo><name type="personal"><namePart type="given">MELVIN M.</namePart><namePart type="family">SCHEINMAN</namePart><affiliation>University of California San Francisco, San Francisco, California</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Science Inc</publisher><place><placeTerm type="text">350 Main Street , Malden , MA 02148‐5018 , USA and 9600 Garsington Road , Oxford OX4 2DQ , UK .</placeTerm></place><dateIssued encoding="w3cdtf">2005-02</dateIssued><edition>Received July 21, 2004; revised August 1, 2004; accepted August 16, 2004.</edition><copyrightDate encoding="w3cdtf">2005</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="figures">1</extent><extent unit="references">53</extent></physicalDescription><abstract lang="en">While Drs. Wolff, Parkinson, and White fully described the syndrome that bears their names in 1930, prior case reports had already described the essentials. Over the ensuing century this syndrome has captivated the interest of anatomists, clinical cardiologists, and cardiac surgeons. Stanley Kent described lateral muscular connections over the atrioventricular (AV) groove, which he felt were the normal AV connections. The normal AV connections were, however, clearly described by His and Tawara. True right‐sided AV connections were initially described by Wood et al., while Öhnell first described left free wall pathways. David Scherf is thought to be the first to describe our current understanding of the pathogenesis of the Wolff‐Parkinson‐White (WPW) syndrome in terms of a reentrant circuit involving both the AV node—His axis as well as the accessory pathway. This hypothesis was not universally accepted and many theories were applied to explain the clinical findings. The basics of our understandings were established by the brilliant work of Pick, Langendorf, and Katz who by using careful deductive analysis of ECGs were able to define the basic pathophysiological processes. Subsequently, Wellens and Durrer applied invasive electrical stimulation to the heart in order to confirm the pathophysiological processes. Sealy and his colleagues at Duke University Medical Center were the first to successfully surgically divide an accessory pathway and ushered in the modern area for curative therapy for these patients. Morady and Scheinman were the first to successfully ablate an accessory pathway (posteroseptal) using high‐energy direct‐current shocks. Subsequently, Jackman, Kuck, Morady, and a number of groups proved the remarkable safety and efficiency of catheter ablation for pathways in all locations using radiofrequency energy. More recently, Gallob et al. first described the gene responsible for a familial form of WPW. The current ability to cure patients with WPW is due to the splendid contributions of individuals from diverse disciplines from throughout the world.</abstract><subject lang="en"><genre>Keywords</genre><topic>historical review</topic><topic>Wolff‐Parkinson‐White syndrome</topic><topic>catheter ablation</topic><topic>cardiac electrosurgery</topic><topic>preexcitation</topic><topic>accessory pathways</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>2005</date><detail type="volume"><caption>vol.</caption><number>28</number></detail><detail type="issue"><caption>no.</caption><number>2</number></detail><extent unit="pages"><start>152</start><end>156</end><total>5</total></extent></part></relatedItem><identifier type="istex">8AF4179AD7FF5D60F05D57E4B7788C05986FA9B3</identifier><identifier type="DOI">10.1111/j.1540-8159.2005.09461.x</identifier><identifier type="ArticleID">PACE9461</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Science Inc</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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